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Solution First, an M-file function must be createdto computethe sum of the squares The following file, called f ssR m,is set up for the power equation: function f = fSSR(a,xm,ym) yp = a(1)*xm^a(2); f = s u m (( y m - y p ) ^ 2 ) ; In commandmode, the datacan be enteredas >> x = t10 20 30 40 50 60 7O B0l; >> y - 125 7A 380 550 610 1220 830 14501; The minimization of the function is then implemented by >> fminsearch (@fSSR,[1, 1] , 25384 r4359 tL x, y)

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The terms subroutines, functions, and methods have all been used to describe the C# event handlers What is the correct term The dual histories of Visual Basic and C# associated with ASPNET have left an unusual legacy of function terminology In ASPNET, a subroutine is a function with no return value A function with a return value is called a function That makes a lot more sense if you re a Visual Basic user, but with C# you generally do not reference a void-typed function as a subroutine The term method is a reference to a C# class function As you have seen, most of the functions that handle the events are clearly part of a class, so the term method is appropriate as well More general terms for functions include operations and routines, but they usually reference the more specific statements within the boundaries of a method Most of the references you will see in this book are to either a function or method, meaning exactly the same thing without regard for whether they include a return value A few subroutines references are added to make those with a Visual Basic background feel comfortable

The besrfit model is therefore a35e F : 25384u1 Both the original transformedfit and the presentversion are displayedi-li'Frg144 Note that althoughthe model coefficientsarevery different,it is difficult to judge which fit is superiorbasedon inspectionof the plot This example illustrateshow different best-fit equations result when fitting the same employingtransformations This model using nonlinearregression versuslinear regression is because former minimizes the residualsof the orisinal datawhereas latter minithe the data mizes the residualsof the transformed

pressure It consists of a triad of hypertension, bradycardia, and slow, irregular breathing elicited by the stimulation of mechanically sensitive regions in the paramedian caudal medulla (Hoff and Reis) Similar pressure-sensitive areas in the upper cervical spinal cord are also implicated in the Cushing response when intraspinal pressure is raised abruptly; a ventral medullary vasodepressor area that acts in the opposite manner has been found in animals The proximate cause of the Cushing response is a mechanical distortion of the lower brainstem, either from a mass in the posterior fossa or, more often, from a large mass in one of the hemispheres or a subarachnoid hemorrhage causing a transmitted pressure that elevates the pressure within the fourth ventricle Often, only the hypertensive component of the reaction occurs, with the systolic blood pressure reaching levels of 200 mmHg, spuriously suggesting the presence of a pheochromocytoma or renal artery stenosis In our experience, the most severe instances of this type of centrally provoked hypertensive syndrome have occurred in children with cerebellar tumors who presented with headache and extreme systolic hypertension Dif culty may arise in differentiating this response from hypertensive encephalopathy, especially from cases that derive from renovascular hypertension, which may likewise be accompanied by headache and papilledema In making a differentiation between these two, it is useful to note that hypertensive encephalopathy is associated with a tachycardia or normal heart rate and that systolic blood pressure levels above 210 mmHg are attained only rarely in the Cushing response In relation to the third type of sympathetic hyperactivity, Pen eld described paroxysms of hypertension, intense diaphoresis, ushed skin, and mydriasis in comatose patients and attributed them to epilepsy (diencephalic seizures), although his original patient had a tumor obstructing the foramen of Munro Many similar types of attacks have been described, additional features being a slight rise in temperature just prior to the paroxysm, cyclic respirations, and shivering Often the bedsheets become soaked and the patient s forehead is covered by beads of sweat Most patients who exhibit such paroxysms are decorticate from traumatic lesions of the deep cerebral white matter or from acute hydrocephalus (the likely explanation of Pen eld s cases); in any case, they are clearly not epileptic We have conceptualized these attacks as the result of the removal of inhibitory in uences on the hypothalamus, creating, in effect, a hypersensitive decorticated autonomic nervous system The condition is perhaps analogous to overactivity of the extensor and opisthotonic posturing mechanisms that appear with little provocation in the same clinical settings Morphine and bromocriptine have been helpful in suppressing but not eliminating the attacks, and beta-adrenergic blockers reduce the hypertension and tachycardia During episodes of intense sympathetic discharge of any type, there are alterations in the ECG, mainly in the ST segments and T waves; in extreme cases, evidence of myocardial damage can be observed The role of direct sympathetic innervation of the heart in producing these myocardial abnormalities is unclear, but in the cases of subarachnoid hemorrhage and traumatic brain injury, the surge in circulating NE and cortisol is postulated as the cause A similar hyperadrenergic mechanism has been proposed to explain sudden death from fright, asthma, status epilepticus, and cocaine overdose Investigations by Schobel and colleagues have suggested that sustained sympathetic overactivity is responsible for the hypertension of pre-eclampsia, which may be considered in some ways as a dysautonomic state Further information on these topics is contained in the reviews by Samuels and by Ropper.