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Determine (a) the mean, (b) median, (c) mode (d) range, (e) standarddeviation, (f) variance,and (g) coefficient of variation (h) Constructa histogramUse a range from 26 to 32 with increments 0-5 of (i) Assuming that the distribution is normal, and that your estimateof the standard deviation is valid, computethe range (that is, the lower and the upper values) that en68Vo<>f readings the Determinewhetherthis compasses is a valid estimatefor the data in this problem 13,1Using the same approachas was empioyed to derive Eqs (1315)and (1316),derivethe least-squales of the fit following model:

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Along with the slopeand intercept,computethe standardenor of the estimate and the correlation coefficient Plot thedata and the regressionline Then repeatthe problem,but regress x versus}-that is, switch the variablesInterpretyour results 136 The following datawas gathered determine relato the tionship betweenpressureand temperature a fixedvolof unteof I kg of nitrogcn The volumeis l0 mr T,'C p,Nlm2 -aO t 2A 0 40 B0 160 6 9 0 0 B1 0 0 9 3 0 0 1 0 , 5 0 0 I 1 , 2 0 0 t 2 9 0 0

function, predominate in certain types of polyneuropathy They occur most frequently in amyloidosis and certain other hereditary small- ber polyneuropathies, especially diabetic, and several congenital types In addition, they are among the main features of an acute autonomic polyneuropathy called pandysautonomia (Young, Adams, et al, Low et al) and can be prominent in some cases of GBS These dysautonomic conditions are described in detail in Chap 26 and later in this chapter Other manifestations of autonomic paralysis are small or medium-sized unreactive pupils that are unusually sensitive to certain drugs (pages 241 and 462); lack of sweat, tears, and saliva; sexual impotence; weak bowel and bladder sphincters with urinary retention or over ow incontinence; and weakness and dilatation of the esophagus and colon As a result of vagal and other parasympathetic dysfunction, the normal variability of heart rate with respiration (sinus arrhythmia) is lost and there may be paralytic ileus or dyscoordinated peristalsis, as well as achlorhydria and hyponatremia Some of these abnormalities are found in diabetic and amyloid polyneuropathy In general these autonomic disturbances correspond to degeneration of unmyelinated bers in the peripheral nerves In any neuropathy involving sensory nerves, there is loss of autonomic function in the same zones as sensory loss This is not true of radicular diseases because the autonomic bers join the spinal nerves more distally Such changes in sweating and cutaneous blood ow may be demonstrated by a number of special tests, as described in Chap 26 Fasciculations, Cramps, and Spasms (See also Chap 55) Fasciculations and cramps, while not uncommon, are typically not prominent features in most polyneuropathies and in this respect there is a difference from diseases of the anterior horn cells where they are important features There are exceptions, however Chronic root compression can lead to fasciculations or painful spasms in the innervated muscles Occasionally one observes a state of mild motor polyneuropathy that, upon recovery, leaves the muscles in a state variably referred to as myokymia, continuous muscular activity, and neuromyotonia as discussed in Chaps 45 and 54 All the affected muscles ripple and quiver and occasionally cramp Use of the muscles increases this activity, and there is a reduction in their contractile ef ciency, which the patient senses as a stiffness and heaviness In some instances this apparently constitutes the entire neuropathic syndrome and may be relieved by carbamazepine or phenytoin Other closely related phenomena are spasms or involuntary movements of the toes and feet The latter, referred to by Spillane and colleagues as the syndrome of painful legs and moving toes, is attributed by Nathan to ectopic discharges in sensory roots, ganglia, or nerves, evoking both pain and organized movements This is one cause of a nocturnal restless leg syndrome, the more common type being idiopathic and not associated with features of peripheral nerve disease Other possible mechanisms for cramps and spasms are ephaptic cross-transmission, segmental hyperactivity from deafferentation, and neuronal sprouting during reinnervation In yet other instances, the muscle activity induces odd postures or slow writhing movements that Jankovic and van der Linden have likened to dystonia The precise pathophysiology of these spontaneous and asynchronous activities of motor neurons is not known Stimulation of a motor nerve, instead of causing a brief burst of action potentials in the muscle, results in a prolonged or dispersed series of potentials lasting several hundred milliseconds Evidently, branched axons involved in.

Figure 6-8

Enrploy the ideal gas law pV - nRT to deterrnine onthe rt basis of this data Note that for the 1aw,I must be expressed in kelvins 137 Beyond the examples in Fig l3ll, there areother models that can be linearized usins transformations FoI example, \) : a^rep1r